aldosterone

Plasma renin activity; Aldosterone; Malignant hypertensive arteriolar nephrosclerosis; Diagnosis.

血浆肾素活性；醛固酮；恶性高血压性小动脉性肾硬化；诊断。

Inhibitors of the renin-angiotensin-aldosterone system may reduce LV mass to a greater extent than other antihypertensive agents.

与其它抗高血压药物相比，肾素-血管紧张素-醛固酮系统抑制剂可以明显减低左室重量。

For the past few years, study of non-classic actions of aldosterone receptor blocker have been hot spot, especially eplerenone.

近年来，醛固酮受体阻滞剂的非传统作用已成为研究热点，尤其是依普利酮的研究。

It blocks that part of the renin-angiotensin-aldosterone hormonal system that narrows (constricts) the peripheral arteries.

一方面，它可以阻断肾素-血管紧缩素-醛固酮的激素系统，该系统会引起周围动脉的狭窄。

Objective To investigate the nursing experience of early diabetic nephropathy by treating with aldosterone receptor antagonist.

目的探讨醛目酮受体拮抗剂治疗早期糖尿病肾病的临床护理体会。

Result: Compared with normal PCR, Gradient PCR could identify the the optimal annealing temperature of aldosterone synthase gene.

结果：与普通PCR相比，梯度PCR能够迅速地确定CYP11B2基因的最佳退火温度。

and action in stimulating the production of aldosterone to promote the reabsorption of sodium and water, further raising the blood pressure.

二是通过刺激分泌醛固酮，引起水钠重吸收增加，使血压进一步升高。

Next, initiate timed urine collection to determine aldosterone levels.

接下来，开始定时留尿以确定醛固酮水平。

Salt and water depletion due to inability to conserve sodium in the TALH or DCT leads to activation of the RAAS and high aldosterone levels.

由于TALH或DCT不能保钠，因而会出现盐和水的丢失，继而会激活RAAS，使醛固酮水平升高。

Lower-sodium diets also boosted levels of the hormones renin and aldosterone, which can raise blood pressure.

低盐饮食也能够增加可以提高血压的肾素水平和醛固酮水平。

Extrarenal volume depletion is a possible reason for low blood pressure, high aldosterone excretion, and potassium loss.

肾外血容量减少是低血压、高醛固酮排泄、血钾丢失的一个可能原因。

The renin-angiotensin-aldosterone system (RAAS) is a feedback system activated with volume depletion.

肾素-血管紧张素-醛固酮系统（RAAS）是一种能被容量丢失激活的反馈控制系统。

If urinary aldosterone levels are high despite volume replacement, there is an abnormal stimulation of aldosterone.

如尽管补充了容量，尿醛固酮水平仍高，即有对醛固酮异常的刺激。

The renin-angiotensin-aldosterone system plays an important role in regulating the circadian rhythms of blood pressure.

肾素-血管紧张素-醛固酮系统在血压昼夜节律的调节中起着关键作用。

Conclusions: The aldosterone level in blood and atrial tissue decreased after AF reversion to sinus rhythm.

结论：房颤恢复窦性心律后血液和心房肌组织中醛固酮水平下降。

Objective: To investigate the effect of aldosterone on the proliferation of cultured myocardial fibroblasts in vitro.

目的：研究醛固酮对培养心肌成纤维细胞增殖的影响。

Exposure to excess aldosterone results in cardiac damage in hypertensive states.

超量的醛固酮作用致使高血压状态下的心脏损害。

The use of aldosterone antagonists for dilated cardiomyopathy improves levels of morbidity and mortality.

在扩张型心肌病治疗上加用醛固酮受体拮抗剂可改善发病率和死亡率。

High levels of aldosterone also enhance potassium and hydrogen exchange for sodium.

高醛固酮水平也能增加钾、氢与钠的交换。

Zona glomerulosa (just beneath the capsule) is the site of aldosterone synthesis and responds to angiotensin and potassium.

球状带（就在被膜下面）是合成醛固酮的地方和负责血管紧张素和钾的代谢。